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"성인호흡곤란증후군 또는 급성호흡곤란증후군에 대한 문헌고찰" 에 대한 레포트 입니다.
간호 서적들을 보며 수정 또 수정을 하며 깔끔하게 작성하였습니다.

본문일부 및 목차

목 차

1. 정의 ········································································································ 1

2. 병태생리 ································································································· 1

3. 원인 ········································································································ 2

4. 증상과 징후 ···························································································· 2

5. 진단 ········································································································ 2

6. 합병증 ····································································································· 2

7. 치료와 간호 ···························································································· 3

참고문헌



성인 호흡곤란 증후군 또는 급성 호흡곤란 증후군
(adult respiratory distress syndrome, ARDS)

1. 정의
갑자기 심한 호흡곤란, 저산소혈증과 미만성 폐침윤으로 과잉탄산증 없이 나타나는 급성 저산소성 호흡부전 증후군이다.

2. 병태생리
1) 손상 또는 삼출기(폐손상 직후 첫 24~48시간, 보통 1~7일 사이 발생)
● 백혈구가 폐의 미세혈관에 부착 → 혈관 내피세포 손상 → 모세혈관막 투과성 증가
● 간질부종 발생 → 간질액이 폐포상피 통과해 폐포 안으로 이동 → 혈액의 산소화 방해
● 상피세포 파괴 → 표면활성물질 생산결핍 → 폐허탈, 폐탄성의 저하, 가스교환 장애, 저산소혈증 발생
● 폐포벽은 죽은 세포, 단백질, 섬유소로 구성된 hyaline 막으로 변화 → 폐섬유화와 허탈로 진행

연관검색어

#ARDS

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